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With enhanced pharmacodynamics, these agents mimic GLP-1 and stimulate insulin release from the pancreas, suppress glucagon secretion (both in a glucose-dependent manner), inhibit gastric emptying, and may lower overall appetite and food intake [17]

Further, the use of exogenous insulins requires unremitting glycaemic monitoring and dose titration to mitigate the risk of hypoglycaemia

Long-term effects and individual variability in response also warrant consideration

By contrast, compounding lacks validated research

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